![]() ![]() Homogeneous, glassy (hyaline) substance, composed Normal components of their walls are replaced by a Affected vessels become thickened, and the Originate from the base of the brain and supply theīasal ganglia, thalamus, deep white matter, and theīrainstem. This pathology affects small penetrating arteries and arterioles that Occur also in old age without these predisposing conditions. Small vessel disease includes atherosclerosis of small arteries but refersĪtreriolosclerosis, a vascular lesion that is seen primarily in hypertension and diabetes but The blood brain barrier is once again sealed. With maturation of new capillaries and glial scar formation, This is completed in approximately 2 months. Undamaged brain proliferate and form a glial scar around the infarct. Macrophage reactionĪppears early and peaks at 3-4 weeks. They ingest the products of degradation of neurons and myelinĪnd are transformed into lipid-laden macrophages. Monocytes from the blood stream enter the infarct throughĭamaged vessels. Neovascularization (which accounts for contrast enhancement) As the core of the infarct disintegrates, endothelialĬells from the periphery proliferate, and capillaries grow into the dead ![]() Of staining and vacuolization of the white matter due to unraveling of myelin,įirst week, there is a transient inflammatory reaction, especiallyĪround blood vessels and in the meninges, due to release of arachidonicĪnd other fatty acids. 24 to 48 hours reveals anoxic neurons, pallor ![]()
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